Molecular mechanisms in relation to cortisol and leucocytes in the pathogenesis of ventricular arrhythmias

Vargova V.¹ , Singh R.B. ² , Chibisov S. ³ , Bawareed A.-O. ⁴ , Isaza A.⁵

World Heart Journal

2

Английский

131-140

Опубликовано

Внешняя ссылка

11

2019

• ¹ PJ Safaric University, Kosice, Slovakia
• ² Halberg Hospital and Research Institute, Moradabad, India
• ³ Faculty of Medicine, RUDN University, Moscow, Russian Federation
• ⁴ Department of Normal Physiology, People’s Friendship University of Russia, Moscow, Russian Federation
• ⁵ Everglade University, Tampa, FL, United States

connexin 43; hydrocortisone; acute coronary syndrome; Article; cardiogenic shock; cardiomyopathy; cardiovascular risk; cause of death; clinical outcome; coronary artery bypass graft; dizziness; DNA methylation; electrophysiology; faintness; fatigue; fibroblast; heart ejection fraction; heart infarction; heart left ventricle failure; heart muscle ischemia; heart ventricle arrhythmia; heart ventricle contraction; histone modification; human; hypothalamus hypophysis adrenal system; immunosurveillance; inflammation; length of stay; leukocyte; macrophage; neutrophil count; pathogenesis; percutaneous coronary intervention; risk factor; stratification; sudden cardiac death

24.12.2019

24.12.2019

https://repository.rudn.ru/ru/records/article/record/55473/